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In this model, CRTC1 was identified as a direct target of miR-184-3p, suggesting that the loss of this miRNA compromises molecular mechanisms essential for HPA axis homeostasis and affective behavior regulation (Xu et al., 2024)

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Hyperbaric oxygen and hyperbaric air treatment result in comparable neuronal death reduction and improved behavioral outcome after transient forebrain ischemia in the gerbil

Overexpression of TFRC contributes to the continuous accumulation of ferrous iron and autophagic lysosome-mediated degradation of ferritin, and promotes lipid peroxidation and cell collapse, resulting in ferroptosis-related cell death [37, 38]